We regret that in the originally published article, the Acknowledgement section was inadvertantly omitted. The corrected section can be found below.
This work was supported by a Research Grant from Keimyung University in 2002 (2002-262).
Apologies for any inconvenience caused.
Received: March 1, 2004
© 2005 Published by Elsevier Inc.
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- A transcriptional factor decoy against AP-1 suppresses TGF-β1-induced type I collagen gene expression in cultured keloid fibroblastsJournal of Dermatological ScienceVol. 37Issue 1
- PreviewDuring wound repair, type I collagen production and deposition in the extracellular matrix is required for the generation of normal physiological connective tissue but these events are regulated on several levels. Keloid occurs when this regulatory process is disrupted in some way (reviewed in Ref. ). TGF-β proteins play an important role in all of these processes and transient TGF-β production may, therefore, promote wound healing and restoration of tissue architecture. However, persistent expression and/or activation of TGF-β may lead to keloid.